Thursday 20 March 2014

Picture Of Lungs Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Picture Of Lungs Biography

(Source google.com)
It has been long established that hostile personality traits are related to cigarette dependency and smoking cessation difficulties. Now UCI researchers have found that in people who have aggressive personalities nicotine triggers significant brain activity in the areas that help control social response, thinking and planning. In turn, non-hostile people showed no brain activity increases at all to nicotine. These findings suggest that some people are born with a predisposition to cigarette addiction and helps explain why quitting for some is practically impossible. It has been long established that hostile personality traits are related to cigarette dependency and smoking cessation difficulties. Now UCI researchers have found that in people who have aggressive personalities nicotine triggers significant brain activity in the areas that help control social response, thinking and planning. In turn, non-hostile people showed no brain activity increases at all to nicotine. These findings suggest that some people are born with a predisposition to cigarette addiction and helps explain why quitting for some is practically impossible. The ability of certain epithelial cells to use active-transport systems, as discussed above, enables them to absorb filtered material, such as glucose from the lumen of the intestine, which can then be circulated to the rest of the body. Cells are also able to endocytose other materials that are necessary for cell growth and signaling. For more information, see transcytosis.  Some epithelial cells, such as the goblet cells, secrete fluids that are necessary for other processes such as digestion, protection, excretion of waste products, lubrication, reproduction, and the regulation of metabolic processes of the body. As part of its excretory role, certain epithelial cells secrete mucus, which lubricate the body cavities (i.e. peritoneum, pericardium, pleura, and tunica vaginalis) and passageways that they line. In the trachea, goblet epithelial cells secrete mucous which provides the lubrication to aid ciliated epithelial cells in sweeping bacteria and dust away from the lungs (Figure 6). In addition, type II alveolar cells excrete pulmonary surfactant, which decreases surface tension, allowing for normal lung function. Figure 6 shows an example of secretory cells in the fallopian tubes. Figure 6. This scanning electron microscope image depicts the ciliated epithelial cells that line the trachea. Some epithelial cells have cilia, which aid in moving substances in the lumen by creating a current via coordinated "sweeping" of the cilia (Figure 6). For instance, ciliated  columnar epithelial cells are instrumental in the movement of the ovum through the Fallopian tubes to the uterus (Figure 7). Study: Take a group of people and, after standardized personality testing, divide them into two groups: a "high" hostile group characterized by more anger, aggression and anxiety as compared to a "low" hostile group. Include smokers and nonsmokers in both the "high" and the "low" hostile groups. Ask everyone to wear nicotine patches and then give them all brain scans. Obtain some interesting results. Nicotine triggered increased brain activity in the "high" hostile group -- whether they were smokers or not. (The "high" hostile smokers did need more nicotine to achieve a response comparable to the "high" hostile nonsmokers.) By contrast, there were no metabolic changes in the brain cells of the low-hostility participants. The results suggest that "high" hostile people respond to nicotine more than "low" hostile people.Conclusion: In people who have aggressive personalities, nicotine triggers significant brain activity in the areas that help control social response, thinking and planning. Comment: Does this mean that hostile people are more likely to start smoking in the first place and can be expected to have a harder time if they then decide to quit smoking. Study: Take a group of people and, after standardized personality testing, divide them into two groups: a "high" hostile group characterized by more anger, aggression and anxiety as compared to a "low" hostile group. Include smokers and nonsmokers in both the "high" and the "low" hostile groups. Ask everyone to wear nicotine patches and then give them all brain scans. Obtain some interesting results. Nicotine triggered increased brain activity in the "high" hostile group -- whether they were smokers or not. (The "high" hostile smokers did need more nicotine to achieve a response comparable to the "high" hostile nonsmokers.) By contrast, there were no metabolic changes in the brain cells of the low-hostility participants. The results suggest that "high" hostile people respond to nicotine more than "low" hostile people. Conclusion: In people who have aggressive personalities, nicotine triggers significant brain activity in the areas that help control social response, thinking and planning.
Comment: Does this mean that hostile people are more likely to start smoking in the first place and can be expected to have a harder time if they then decide to quit smoking? Pulmonary/Critical Care Medicine at Cedars-Sinai Medical Center and Professor of Medicine at UCLA. UCI study reveals why some people may be 'born to smoke' Nicotine study provides first results showing personality traits, brain activity and cigarette addiction link Why are some people hopelessly addicted to cigarettes, while others seemingly can quit at will? A UC Irvine College of Medicine study reveals for the first time the underlying brain mechanisms that link personality traits to nicotine addiction.

Picture Of Lungs Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Picture Of Lungs Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Picture Of Lungs Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Picture Of Lungs Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Picture Of Lungs Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Picture Of Lungs Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Picture Of Lungs Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Picture Of Lungs Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Picture Of Lungs Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Picture Of Lungs Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Picture Of Lungs Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Picture Of Lungs Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Picture Of Lungs Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Picture Of Lungs Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Picture Of Lungs Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Lung Desease Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Lung Desease Biography

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Chronic obstructive pulmonary disease (COPD), which includes emphysema, is an example of an obstructive lung disease where the alveolae rupture, thus causing air to be retained in the lungs and hence limiting the available space during inhalation. Asthma is an example of an obstructive lung disease (and of an inflammatory lung disease). It is a disease in which muscles of bronchi contract, making it difficult for air to reach the lungs. One treatment of asthma is to use an inhaler which contains a drug to relax muscles of bronchi. Asthma is a difficulty in breathing causing wheezing due to inflammation of bronchi and bronchioles, this causes a restriction in the airflow into the alveoli. Asthma attacks can be brought on by triggers, such as air pollution, tobacco smoke, factory fumes, cleaning solvents, infections, pollens, dust, foods, cold air, exercise, chemicals and medications. Triggers are highly individual and may not be related to allergens. Many asthmatics are not allergic to common allergens such as mold. Restrictive lung diseases are a category of respiratory disease characterized by a loss of lung compliance, causing incomplete lung expansion and increased lung stiffness, such as in infants with respiratory distress syndrome.
Infections can affect any part of the respiratory system. They are traditionally divided into upper respiratory tract infections and lower respiratory tract infections. The most common upper respiratory tract infection is the common cold. However, infections of specific organs of the upper respiratory tract such as sinusitis, tonsillitis, otitis media, pharyngitis and laryngitis are also considered upper respiratory tract infections. The most common lower respiratory tract infection is pneumonia, an infection of the lungs which is usually caused by bacteria, particularly Streptococcus pneumoniae in Western countries. Worldwide, tuberculosis is an important cause of pneumonia. Other pathogens such as viruses and fungi can cause pneumonia for example severe acute respiratory syndrome and pneumocystis pneumonia. A pneumonia may develop complications such as a lung abscess, a round cavity in the lung caused by the infection, or may spread to the pleural cavity. Malignant tumors of the respiratory system, particularly primary carcinomas of the lung, are a major health problem responsible for 15% of all cancer diagnoses and 30% of all cancer deaths.The majority of respiratory system cancers are attributable to smoking tobacco. The major histological types of respiratory system cancer are: In addition, since many cancers spread via the bloodstream and the entire cardiac output passes through the lungs, it is common for cancer metastases to occur within the lung. Breast cancer may invade directly through local spread, and through lymph node metastases. After metastasis to the liver, colon cancer frequently metastasizes to the lung. Prostate cancer, germ cell cancer and renal cell carcinoma may also metastasize to the lung. Treatment of respiratory system cancer depends on the type of cancer. Surgical removal of part of a lung (lobectomy, segmentectomy, or wedge resection) or of an entire lung pneumonectomy), along with chemotherapy and radiotherapy, are all used. The chance of surviving lung cancer depends on the cancer stage at the time the cancer is diagnosed, and to some extent on the histology, and is only about 14-17% overall. In the case of metastases to the lung, treatment can occasionally be curative but only in certain, rare circumstances.
Benign tumors are relatively rare causes of respiratory disease. Examples of benign tumors are: Pleural cavity diseases include pleural mesothelioma which are mentioned above. A collection of fluid in the pleural cavity is known as a pleural effusion. This may be due to fluid shifting from the bloodstream into the pleural cavity due to conditions such as congestive heart failure and cirrhosis. It may also be due to inflammation of the pleura itself as can occur with infection, pulmonary embolus, tuberculosis, mesothelioma and other conditions. A pneumothorax is a hole in the pleura covering the lung allowing air in the lung to escape into the pleural cavity. The affected lung “collapses” like a deflated balloon. A tension pneumothorax is a particularly severe form of this condition where the air in the pleural cavity cannot escape, so the pneumothorax keeps getting bigger until it compresses the heart and blood vessels, leading to a life threatening situation.

Lung Desease Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker


Lung Desease Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Lung Desease Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker


Lung Desease Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker


Lung Desease Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker


Lung Desease Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker


Lung Desease Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker


Lung Desease Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker


Lung Desease Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker


Lung Desease Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker


Lung Desease Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker


Lung Desease Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker


Lung Desease Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Lung Desease Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Lung Sound Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Lung Sound Biography

(Source google.com)
Lung sounds, breath sounds, or Respiratory sounds refer to the specific sounds generated by the movement of air through the respiratory system. These may be easily audible or identified through auscultation of the respiratory system through the lung fields. with a stethoscope. These include normal breath sounds and adventitious or "added" sounds such as crackles, wheezes, pleural friction rubs, stertor and stridor. Pectoriloquy, egophony and bronchophony are a tests of auscultation. For example, in whispered pectoriloquy the patient is asked to whisper - typically a two syllable number as the clinician listens over the lung fields. The whisper is not normally heard over the lungs, but if heard may be indicative of pulmonary consolidation in that area. This is because sound travels differently through denser (fluid or solid) media than the air that should normally be predominant in lung tissue. Description and classification of the sounds usually involves auscultation of the inspiratory and expiratory phases of the breath cycle, noting both the pitch (typically described as low, medium or high) and intensity (soft, medium, loud or very loud) of the sounds heard. "Rhonchi" and "rales" are obsolete terminology whose use in the literature has been variable. The terms wheeze and crepitation have replaced them.
Breath sounds can be classified into two categories, either NORMAL or ABNORMAL (adventitious).  Breath sounds originate in the large airways where air velocity and turbulence induce vibrations in the airway walls.  These vibrations are then transmitted through the lung tissue and thoracic wall to the surface where they may be heard readily with the aid of a stethescope.  Normal breath sound production is directly related to air flow velocity and airway lumen architecture.  Air flow velocity is primarily determined by pulmonary ventilation and TOTAL cross sectional airway area at any given level in the lungs. It is a common misconception that air moving through terminal bronchioles (airways with a diameter <2 mm) and alveoli also contribute to breath sounds.  This is incorrect as the air velocity at this level is too slow (very large total cross sectional area) to produce significant turbulence and sound waves.  However, terminal airway and alveolar disease does modify the breath sounds heard at the surface by either increasing or decreasing the sound transmission through the diseased tissue.  Thus, the sounds that are heard at the periphery of the lung are produced in more central (hilar) regions and are altered in intensity and tonal quality as they pass through pulmonary tissue to the periphery.
Bronchial breath sounds consist of a full inspiratory and expiratory phase with the inspiratory phase usually being louder.  They are normally heard over the trachea and larynx.  Bronchial sounds are not normally heard over the thorax in resting animals.  They may be heard over the hilar region in normal animals that are breathing hard (i.e. after exercise).  Otherwise, bronchial sounds heard over the thorax suggest lung consolidation and pulmonary disease.  Pulmonary consolidation results in improved transmission of breath sounds originating in the trachea and primary bronchi that are then heard at increased intensity over the thorax.
Bronchovesicular breath sounds consist of a full inspiratory phase with a shortened and softer expiratory phase.  They are normally heard over the hilar region in most resting animals and should be quieter than the tracheal breath sounds.  However, in sheep, goats, llamas, and alpacas, they may be heard throughout the full lung field and are often louder than tracheal breath sounds.  Increased intensity of bronchovesicular sounds is most often  associated with increased ventilation or pulmonary consolidation. Vesicular breath sounds consist of a quiet, wispy inspiratory phase followed by a short, almost silent expiratory phase.  They are heard over the periphery of the lung field.  As stated earlier, these sounds are NOT produced by air moving through the terminal bronchioles and alveoli but rather are the result of attenuation of breath sounds produced in the bronchi at the hilar region of the lungs.  These sounds may be absent or silent in the periphery of normal resting animals.  They are highly variable in intensity depending on the species, ventilation, and body condition.  Increased intensity may be associated with pulmonary consolidation.

Lung Sound Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker


Lung Sound Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker


Lung Sound Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker


Lung Sound Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker


Lung Sound Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker


Lung Sound Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker


Lung Sound Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker


Lung Sound Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker


Lung Sound Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker


Lung Sound Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker


Lung Sound Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker


Lung Sound Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker


Lung Sound Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker


Lung Sound Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker


Lung Sound Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker


Lung Sound Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker


Lung Sound Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Lung Structure Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Lung Structure Biography

(Source google.com)
The avian respiratory system delivers oxygen from the air to the tissues and also removes carbon dioxide. In addition, the respiratory system plays an important role in hermoregulation (maintaining normal body temperature). The avian respiratory system is different from that of other vertebrates, with birds having relatively small lungs plus nine air sacs that play an important role in respiration (but are not directly involved in the exchange of gases). Pulmonary air-sac system of a Common Teal (Anas crecca). a. Latex injection (blue) highlighting the location of air sacs. b, Main components of the avian flow-through system. Abd, abdominal aire sac; Cdth, caudal thoracic air sac; Cl, clavicular air sac; Crth, cranial thoracic air sac; Cv, cervical air sac; Fu, furcula; Hu, humerus; Lu, lung; Lvd, lateral vertebral diverticula;  Pv, pelvis; and Tr, trachea (From: O'Connor and Claessens 2005).
The air sacs permit a unidirectional flow of air through the lungs. Unidirectional flow means that air moving through bird lungs is largely 'fresh' air & has a higher oxygen content. In contrast, air flow is 'bidirectional' in mammals, moving back and forth into and out of the lungs. As a result, air coming into a mammal's  lungs is mixed with 'old' air (air that has been in the lungs for a while) & this 'mixed air' has less oxygen. So, in bird lungs, more oxygen is available to diffuse into the blood (avian respiratory system). The air sacs permit a unidirectional flow of air through the lungs. Unidirectional flow means that air moving through bird lungs is largely 'fresh' air & has a higher oxygen content. In contrast, air flow is 'bidirectional' in mammals, moving back and forth into and out of the lungs. As a result, air coming into a mammal's  lungs is mixed with 'old' air (air that has been in the lungs for a while) & this 'mixed air' has less oxygen. So, in bird lungs, more oxygen is available to diffuse into the blood (avian respiratory system). Bird-like respiratory systems in dinosaurs -- A recent analysis showing the presence of a very bird-like pulmonary, or lung, system in predatory dinosaurs provides more evidence of an evolutionary link between dinosaurs and birds. First proposed in the late 19th century, theories about the animals' relatedness enjoyed brief support but soon fell out of favor. Evidence gathered over the past 30 years has breathed new life into the hypothesis. O'Connor and Claessens (2005) make clear the unique pulmonary system of birds, which has fixed lungs and air sacs that penetrate the skeleton, has an older history than previously realized. It also dispels the theory that predatory dinosaurs had lungs similar to living reptiles, like crocodiles.
The avian pulmonary system uses "flow-through ventilation," relying on a set of nine flexible air sacs that act like bellows to move air through the almost completely rigid lungs. Air sacs do not take part in the actual oxygen exchange, but do greatly enhance its efficiency and allow for the high metabolic rates found in birds. This system also keeps the volume of air in the lung nearly constant. O'Connor says the presence of an extensive pulmonary air sac system with flow-through ventilation of the lung suggests this group of dinosaurs could have maintained a stable and high metabolism, putting them much closer to a warm-blooded existence. "More and more characteristics that once defined birds--feathers, for example--are now known to have been present in dinosaurs, so, many avian features may really be dinosaurian," said O'Connor. A portion of the air sac actually integrates with the skeleton, forming air pockets in otherwise dense bone. The exact function of this skeletal modification is not completely understood, but one explanation theorizes the skeletal air pockets evolved to lighten the bone structure, allowing dinosaurs to walk upright and birds to fly.

Lung Structure Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Lung Structure Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Lung Structure Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Lung Structure Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Lung Structure Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Lung Structure Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Lung Structure Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Lung Structure Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Lung Structure Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Lung Structure Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Lung Structure Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Lung Structure Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Lung Structure Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Lung Structure Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker

Lung Structure Lungs Diagram of a Smoker after Smoking Cancer Anatomy And Heart Drawing Images AFter Smoking Wee of a Weed Smoker